A New View Duchenne Muscular Dystrophy

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Leonora Grinio Ph
Leonora Grinio Ph

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GJSFR Volume 21 Issue C4

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Duchenne Muscular Dystrophy is the result of mutation gene-dystrophine, productprotein-dystrophin presents in organism as the complexes proteins placing everywhere, their role unclear. Suppose all dystrophine complexes work as one functional System D, thanks signal ability complexes. Suppose the System D the ancient and appeared when the gene dystrophin-utrophin divided into two genes dystrophin and utrophin at early vertebrates. Perfect this System made the gene the longest in human genome. The surprising activity creat inkinase-21-23 000 ME, found by author, make to think of the damage much membranesdamage System. Destroy System D is beginning of the disease, finishing apoptosis-general destructive factor. Two factors determinate the disease-damage the system D and apoptosis.

Funding

No external funding was declared for this work.

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The authors declare no conflict of interest.

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No ethics committee approval was required for this article type.

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Not applicable for this article.

Leonora Grinio Ph. 2021. \u201cA New View Duchenne Muscular Dystrophy\u201d. Global Journal of Science Frontier Research - C: Biological Science GJSFR-C Volume 21 (GJSFR Volume 21 Issue C4): .

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Duchenne Muscular Dystrophy, genetic disorder affecting muscles, analyzed in this academic journal article.
Journal Specifications

Crossref Journal DOI 10.17406/GJSFR

Print ISSN 0975-5896

e-ISSN 2249-4626

Keywords
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GJSFR-C Classification: FOR Code: 279999
Version of record

v1.2

Issue date

October 4, 2021

Language

English

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Duchenne Muscular Dystrophy is the result of mutation gene-dystrophine, productprotein-dystrophin presents in organism as the complexes proteins placing everywhere, their role unclear. Suppose all dystrophine complexes work as one functional System D, thanks signal ability complexes. Suppose the System D the ancient and appeared when the gene dystrophin-utrophin divided into two genes dystrophin and utrophin at early vertebrates. Perfect this System made the gene the longest in human genome. The surprising activity creat inkinase-21-23 000 ME, found by author, make to think of the damage much membranesdamage System. Destroy System D is beginning of the disease, finishing apoptosis-general destructive factor. Two factors determinate the disease-damage the system D and apoptosis.

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A New View Duchenne Muscular Dystrophy

Leonora Grinio Ph
Leonora Grinio Ph

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