Diabetes and Organ Dysfunction in the Developing and Developed World

Ian James Martins
Ian James Martins * §
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§ Edith Cowan University Edith Cowan University

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Diabetes and Organ Dysfunction in the Developing and Developed World

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Abstract

Induction of global organ disease has become important with the events related to diabetes in both the developed and developing world. Type 2 diabetes and peripheral organ disease are connected to Type 3 diabetes that involves the brain early in life associated with brain diseases (stroke, dementia, Alzheimer’s disease).The incidence of diabetes has been predicted to increase to 21% by 2050. In various continents the rise in the global diabetes epidemic has been associated with diseases of various organ diseases related to obesity, diabetes and neurodegenerative diseases (Parkinson’s disease and Alzheimer’s disease). Nutritional therapy has become of central importance as early nutritional therapy may delay organ disease and aging.

References

50 Cites in Article
  1. I Martins (2014). The Global Obesity Epidemic is Related to Stroke, Dementia and Alzheimer's disease.
  2. Dirk Sander,Kerstin Sander,Holger Poppert (2008). Review: Stroke in type 2 diabetes.
  3. Ian Martins,Wei Lim,Andrea Wilson,Simon Laws,Ralph Martins (2013). The acceleration of aging and Alzheimer’s disease through the biological mechanisms behind obesity and type II diabetes.
  4. I Martins (2013). Increased Risk for Obesity and Diabetes with Neurodegeneration in Developing Countries.
  5. I Martins,A Wilson,W Lim,S Laws,S Laws (2012). Sirtuin 1 mediates the obesity induced risk of common degenerative.
  6. I Martins (2014). Nutritional and Genotoxic Stress Contributes to Diabetes and Neurodegenerative Diseases such as Parkinson's and Alzheimer's Diseases.
  7. J Yoshino,S Klein (2013). A novel link between circadian clocks and adipose tissue energy metabolism.
  8. I Martins,R Creegan,W Lim,R Martins (2013). Molecular insights into appetite control and neuroendocrine disease as risk factors for chronic diseases in Western countries. Special Issue. Molecular Mechanisms Involved in Inflammation and Insulin Resistance in Chronic Diseases and Possible Interventions.
  9. Ian Martins,Rhona Creegan (2014). Links between Insulin Resistance, Lipoprotein Metabolism and Amyloidosis in Alzheimer’s Disease.
  10. Ian Martins (2014). Induction of NAFLD with Increased Risk of Obesity and Chronic Diseases in Developed Countries.
  11. Dirk Sander,Kerstin Sander,Holger Poppert (2008). Review: Stroke in type 2 diabetes.
  12. J Janson,T Laedtke,J Parisi,P O'brien,R Petersen,P Butler (2004). Increased risk of type 2 diabetes in Alzheimer disease.
  13. H Liang,Y Zhong,S Zhou,Q Li (2011). Palmitic acid-induced apoptosis in pancreatic β-cells is increased by liver X receptor agonist and attenuated by eicosapentaenoate.
  14. K Maedler,G Spinas,D Dyntar,W Moritz,N Kaiser,Marc Donath (2001). Distinct Effects of Saturated and Monounsaturated Fatty Acids on β-Cell Turnover and Function.
  15. C Wang,Y Guan,Yang (2010). Cytokines in the Progression of Pancreatic β-Cell Dysfunction.
  16. Louis Feurino,William Fisher,Uddalak Bharadwaj,Qizhi Yao,Changyi Chen,Min Li (2006). Current Update of Cytokines in Pancreatic Cancer: Pathogenic Mechanisms, Clinical Indication, and Therapeutic Values.
  17. A Ropero,P Alonso‐magdalena,E García‐garcía,C Ripoll,E Fuentes,A Nadal (2008). Bisphenol‐A disruption of the endocrine pancreas and blood glucose homeostasis.
  18. Jichun Yang,Yujing Chi,Brant Burkhardt,Youfei Guan,Bryan Wolf (2010). Leucine metabolism in regulation of insulin secretion from pancreatic beta cells.
  19. A Brouwer,P Carroll,I Atwater (1991). Effects of leucine on insulin secretion and beta cell membrane potential in mouse islets of Langerhans.
  20. Walter Zawalich,Kathleen Zawalich (1997). Influence of Pyruvic Acid Methyl Ester on Rat Pancreatic Islets.
  21. M Macdonald (1995). Feasibility of a mitochondrial pyruvate malate shuttle in pancreatic islets. Further implication of cytosolic NADPH in insulin secretion.
  22. Daniel Bakowski,Anant Parekh (2007). Regulation of Store-Operated Calcium Channels by the Intermediary Metabolite Pyruvic Acid.
  23. Xiaocun Sun,Michael Zemel (2007). Leucine and Calcium Regulate Fat Metabolism and Energy Partitioning in Murine Adipocytes and Muscle Cells.
  24. K Dunn,M Nelson (2010). Calcium and diabetic vascular dysfunction. Focus on "Elevated Ca2+ sparklet activity during acute hyperglycemia and diabetes in cerebral arterial smooth muscle cells.
  25. Anant Parekh (2000). Calcium signaling and acute pancreatitis: Specific response to a promiscuous messenger.
  26. Katherine Whalley (2011). Calcium sets the tempo.
  27. I Martins (2013). Appetite dysregulation and obesity in Western Countries.
  28. Emma Jones Acquisition Editor LAP LAMBERT Academic Publishing is a trademark of.
  29. Carl Lavie,Mandeep Mehra,Richard Milani (2005). Obesity and heart failure prognosis: paradox or reverse epidemiology?.
  30. B Mathew (2008). Obesity: Effects on Cardiovascular Disease and its Diagnosis.
  31. L Guarente (2007). Sirtuins in aging and disease.
  32. I Martins (2015). Unhealthy Nutrigenomic Diets Accelerate NAFLD and Adiposity in Global communities.
  33. John Bullen,Susann Bluher,Theodoros Kelesidis,Christos Mantzoros (2007). Regulation of adiponectin and its receptors in response to development of diet-induced obesity in mice.
  34. O Al-Attas,N Al-Daghri,K Al-Rubeaan,Silva Da,S Sabico (2009). Changes in endotoxin levels in T2DM subjects on anti-diabetic therapies.
  35. María Vaccaro,Ezequiel Calvo,Angela Suburo,Daniel Sordelli,Gustavo Lanosa,Juan Iovanna (2000). Lipopolysaccharide Directly Affects Pancreatic Acinar Cells.
  36. Ian Martins (2015). LPS Regulates Apolipoprotein E and A<i>β</i> Interactionswith Effects on Acute Phase Proteins and Amyloidosis.
  37. I Martins,V Gupta,A Wilson,S Fuller,R Martins (2014). Interactions Between Apo E and Amyloid Beta and their Relationship to Nutriproteomics and Neurodegeneration.
  38. Carol Murray,Donal Skelly,Colm Cunningham (2011). Exacerbation of CNS inflammation and neurodegeneration by systemic LPS treatment is independent of circulating IL-1β and IL-6.
  39. Lee,Magkos Mantzoros,C Kang,E (2011). Effects of leptin and adiponectin on pancreatic β-cell function.
  40. L Wang,L Li,X Ran,M Long,M Zhang (2013). Lipopolysaccharides reduce adipogenesis in 3T3-L1 adipocytes through activation of NF-κB pathway and downregulation of AMPK expression.
  41. C Vatier,S Kadiri,A Muscat,C Chapron,J Capeau,B Antoine (2012). Visceral and subcutaneous adipose tissue from lean women respond differently to lipopolysaccharide-induced alteration of inflammation and glyceroneogenesis.
  42. F Wang,M Herrington,J Larsson,J Permert (2003). The relationship between diabetes and pancreatic cancer.
  43. Maureen Morrison (2012). Pancreatic Cancer and Diabetes.
  44. Martin Heni,Jürgen Machann,Harald Staiger,Nina Schwenzer,Andreas Peter,Fritz Schick,Claus Claussen,Norbert Stefan,Hans‐ulrich Häring,Andreas Fritsche (2010). Pancreatic fat is negatively associated with insulin secretion in individuals with impaired fasting glucose and/or impaired glucose tolerance: a nuclear magnetic resonance study.
  45. Katherine Pinnick,Stephan Collins,Constantine Londos,Dominique Gauguier,Anne Clark,Barbara Fielding (2008). Pancreatic Ectopic Fat Is Characterized by Adipocyte Infiltration and Altered Lipid Composition.
  46. Shi-Ping Ding,J Li,Jin (2003). A mouse model of severe acute pancreatitis induced with caerulein and lipopolysaccharide.
  47. R Taira,S Yamaguchi,K Shimizu,K Nakamura,T Ayabe,Taira (2015). Bacterial cell wall components regulate adipokine secretion from visceral adipocytes.
  48. Shigeyuki Obara,Sumio Akifusa,Wataru Ariyoshi,Toshinori Okinaga,Michihiko Usui,Keisuke Nakashima,Tatsuji Nishihara (2014). Pyroglutamated Apelin-13 Inhibits Lipopolysaccharide-Induced Production of Pro-Inflammatory Cytokines in Murine Macrophage J774.1 Cells.
  49. M Okamoto,M Ohara-Imaizumi,N Kubota,S Hashimoto,K Eto (2008). Adiponectin induces insulin secretion in vitro and in vivo at a low glucose concentration.
  50. I Martins (2015). Nutritional diets accelerate amyloid beta metabolism and prevent the induction of chronic diseases and Alzheimer's disease.
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Funding

No external funding was declared for this work.

Conflict of Interest

The authors declare no conflict of interest.

Ethical Approval

No ethics committee approval was required for this article type.

Data Availability

Not applicable for this article.

How to Cite This Article

Ian James Martins. 2015. "Diabetes and Organ Dysfunction in the Developing and Developed World". Global Journal of Medical Research - F: Diseases GJMR-F Volume 15 (GJMR Volume 15 Issue F1).

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Journal Specifications

Crossref Journal DOI 10.17406/gjmr

Print ISSN 0975-5888

e-ISSN 2249-4618

Keywords
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Classification
GJMR-F Classification NLMC Code: WD 200
  • Submission ReceivedApril 6, 2015
  • Peer Review Double Blind
    Handling Editor
  • Accepted May 4, 2015
  • Published May 15, 2015
Version of record

v1.2

Issue date
March 24, 2015

Language
English
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Diabetes and Organ Dysfunction in the Developing and Developed World

Ian Martins
Ian Martins Edith Cowan University
  • Submission ReceivedApril 6, 2015
  • Peer Review Double Blind
    Handling Editor
  • Accepted May 4, 2015
  • Published May 15, 2015